Response Inhibition: Knowing No and Still Acting.
Response inhibition is a measurable stopping process carried by a broad brain network; weaker stopping accompanies several disorders and heavy use of some substances, and training it directly produces small effects on eating and health behaviour. Here is what the science actually says, and what to do with it.
01The Opening
The speed of the stopping process determines which actions are cancelled
In an experience-sampling study, in which participants report their states as they occur, 205 adults logged 7,827 episodes of desire over one week.[5] Most desires caused no conflict. Those that clashed with a goal usually prompted an attempt to resist, and resistance succeeded unevenly.[5] This article examines one measurable part of that gap between intention and action: the ability to cancel an action that has already started, which researchers call response inhibition. Response inhibition is the part of self-control whose speed a laboratory can estimate in milliseconds.
Laboratories measure it with two tasks. In the go/no-go task, a person presses a key on most trials and withholds the press when a designated stimulus appears; a press on a withhold trial counts as a commission error.[2] In the stop-signal task, every trial begins with a go cue, and on some trials a second signal tells the person to cancel a response that is already under way.[6][7]
The stop-signal task makes the article's subject concrete. Participants know the rule on every trial. Yet at the delay the task settles on, they fail to stop on about half of stop trials, because the procedure adjusts the delay until that happens.[7] In this task, knowing the rule is constant; whether the action is cancelled depends on how fast the stopping process runs relative to the action.[8]
ADHD, a disorder with a replicated stopping deficit, has a pooled worldwide prevalence of 5.29% in children and adolescents, from 102 studies covering 171,756 subjects. Most of the variation between studies came from differences in their methods.[9]
In the stop-signal task the rule is known on every trial; the speed of the stopping process determines which actions are cancelled.
Response inhibition is one form of a broader capacity. One review places inhibition among three core executive functions, the control processes that also include working memory and cognitive flexibility. It divides inhibition into response inhibition, which covers resisting temptation, and interference control, which covers resisting distraction.[10] A dual-systems account describes behaviour as the joint product of an impulsive system and a reflective system, with self-control as the reflective system overriding impulse.[11]
The word inhibition covers more than one process. A latent-variable study of 220 adults found that overriding a prepotent response, one made strong by habit or frequency, was closely related to resisting distractors, and that both were unrelated to resisting intrusion from information learned earlier.[12] The authors concluded that the term inhibition had been overextended.[12] A taxonomy from developmental psychopathology also separates several kinds of inhibition, including motor inhibition.[13]
This article therefore uses response inhibition in its narrow sense: stopping a motor action, as measured by the stop-signal and go/no-go tasks. A slow stop on a key press is a specific measurement, and results from it do not transfer automatically to every behaviour people call self-control.
Inhibition is several related processes, not one trait; this article follows the one that cancels an action already started.
Two older lines of self-control research need separating from this one. The first comes from delay of gratification research, in which preschoolers who waited longer for a larger reward developed into more cognitively and socially competent adolescents, and attentional strategies helped them wait.[14] A later conceptual replication found the link to achievement at age 15 about half the original size, and controls for family background and early ability cut it by a further two-thirds.[15]
The second treats self-control as a limited resource that one act of restraint drains for the next, an effect called ego depletion. An earlier meta-analysis of 83 studies backed it.[16] A preregistered replication across 23 laboratories with 2,141 participants then found d = 0.04, with a 95% confidence interval from -0.07 to 0.15 that includes zero.[17] That test measured performance on a second task after a first, not stopping; the companion deep dive on ego depletion research covers it in full.
Neither result decides anything about response inhibition. The delay studies measured waiting, and the depletion studies measured performance across sequential tasks. Response inhibition research asks a narrower question with a laboratory task: how fast a started action can be cancelled, and which brain systems do the cancelling.
Two older self-control findings have shrunk or failed on replication; the stopping literature is narrower and is judged here on its own data.
02The Mechanism
The Response Inhibition Race: How the Brain Cancels an Action
In the stop-signal task, people know the rule on every trial and still fail about half the stops. The task is built that way.
In the stop-signal task a stop process races a go process, and a network spanning right prefrontal cortex and the basal ganglia carries the stop; the exact subcortical route is disputed.
The standard account of response inhibition is a race. In the race model, a go process starts when the action is triggered and a stop process starts when the stop signal appears; whichever finishes first determines whether the response is made.[8] The model treats the two processes as independent. That assumption lets researchers estimate a quantity no one can observe directly: the time the stop process takes.[8][6]
That quantity is the stop-signal reaction time (SSRT), the estimated latency of stopping. It is computed as the go reaction time minus the average stop-signal delay, the interval between the go cue and the stop signal.[6] As an illustration of the arithmetic only, a go time of 300 milliseconds and an average delay of 100 milliseconds give an SSRT of 200 milliseconds. A longer SSRT means slower stopping.
Consensus guidance sets the conditions that make SSRT estimates comparable. It recommends stop signals on 25% of trials, a tracking staircase that moves the delay in steps such as 50 milliseconds until stopping succeeds on about half of stop trials, and at least 50 stop trials for a group-level estimate.[7] These rules matter because SSRT is an estimate, and its accuracy depends on the number and design of the stop trials.[7]
SSRT is an estimate derived from a model, not a direct reading, and its quality depends on how the task was run.
The best-known neural account places a brake in the frontal lobe. It holds that a sector of the right inferior frontal gyrus (rIFG), on the lower side of the right frontal lobe, works with the basal ganglia to stop actions completely or to pause them partly, when a stop signal or other salient event appears or when a goal requires it.[18] Imaging that traced white-matter connections, a method called diffusion tractography, found a tract linking right inferior frontal cortex to the region of the subthalamic nucleus (STN), a small basal ganglia structure.[19] Functional imaging in the same work showed activation in the pre-supplementary motor area (preSMA), in front of the motor areas, as well as in the rIFG and STN.[19]
The proposed fast route from frontal cortex to the STN is the hyperdirect pathway; a slower indirect pathway runs from frontal cortex through the striatum and pallidum. In one functional MRI study of 20 people, a model that combined both routes explained stop-trial activity best, and connection strength was highest on trials where stopping succeeded.[20]
Timing data come from rare recordings inside the human brain. In electrocorticography, electrodes placed on the brain surface of patients record activity directly. In four patients, an rIFG response in the beta band, about 16 Hz, was larger for successful than failed stops and occurred 100 to 250 milliseconds after the stop signal.[21] That window is a response latency measured in four people, not a deadline that stopping outside the laboratory must meet.
The proposed circuit links right frontal cortex, the preSMA and the subthalamic nucleus, and its human timing data come from four patients.
The most detailed timing comes from rats. Recording single neurons during a rat stop-signal task, one study found that STN neurons responded to stop cues at short latency whether or not the action was cancelled, while neurons in the substantia nigra pars reticulata (SNr), a basal ganglia output nucleus, responded only when cancellation succeeded.[22] The authors proposed a race between this stop signal and rising input from the striatum, with a point of no return after which the action escapes.[22]
A later account separates stopping into two steps. The pause-then-cancel model proposes a pause process and a separate cancel process in place of a single stop process.[23] A related theory holds that unexpected events of many kinds recruit the same stopping network and produce global motor suppression, a suppressive effect on basal ganglia output that is not limited to the action being stopped.[24]
Rat lesion and drug studies add detail. Lesions of orbitofrontal cortex slowed SSRT, while STN lesions left SSRT unchanged but reduced stopping accuracy at every stop-signal delay.[25] Reviews of the rat work place the STN at the centre of behavioural inhibition.[26] In healthy volunteers, blocking noradrenaline reuptake changed stop-signal performance.[27] The figure below combines these sources, and its pathway links rest on animals, a handful of patients and small imaging samples.
Rat recordings underlie a pause-then-cancel account with a point of no return; human data for that sequence are sparse.
A stop cue starts a second process. The stop process races the go process; the first to finish decides the response.
Frontal cortex and basal ganglia carry the stop. Right IFG and preSMA activate on stop trials; in rats, STN and SNr activity separates successful from failed stops.
A late stop loses the race. If the stop finishes after the go process passes the point of no return, the action escapes.
A schematic of the race model and the proposed circuit; pathway links come from rodent recordings and small human studies and are disputed in pooled imaging. In the race model the stop process runs against the go process, and the first to finish decides the response. SSRT = go reaction time minus mean stop-signal delay. Illustrative arithmetic only: 300 ms minus 100 ms = 200 ms.
Diagram
100 to 250 ms after a stop signal, the window in which a right inferior frontal response was larger on successful than on failed stops, in intracranial recordings from four patients[21]
Intracranial recording · 4 patients · response latency, not a stopping deadline
The circuit account holds at the level of networks and is contested at the level of single regions. A meta-analysis of 225 functional MRI studies, comprising 323 experiments, found right inferior frontal gyrus, insula, middle cingulate and paracingulate cortex and superior parietal cortex active across three kinds of inhibition: resolving interference, withholding an action and cancelling one.[28] The core systems were fronto-parietal and ventral attention networks, not a single brake region.[28]
Other findings point the same way. One imaging study argues that rIFG activity reflects attention to salient cues as well as inhibition.[29] Twelve patients with damage to the left inferior frontal gyrus made more go/no-go errors than controls, most when withhold trials were rare, which qualifies the right-sided view; patients with orbitofrontal damage were unimpaired.[30]
The subcortical route is disputed most directly. A pooled analysis of five functional MRI datasets found no evidence of hyperdirect or indirect pathway activity on successful stops and found large subcortical activity on failed stops.[31] The pathway rests on rodent recordings, tractography and small human studies, and pooled human imaging has not detected it.[22][19][31] A recent review judges the circuit to be on increasingly firm ground and asks whether it generalises to speech, gait, driving and conditions such as stuttering, tics and freezing of gait.[32]
The stopping network is broad and shared across kinds of inhibition; the fast subcortical route rests on animal and small human studies and has not appeared in pooled human imaging.
03Evidence
What Response Inhibition Deficits Predict, and What They Do Not
01At a glance
The Evidence at a Glance · 04 Findings
What the Stopping Research Found
Four pooled results: a stopping delay in ADHD, inhibition deficits in heavy use of some substances and not others, and two small training effects on eating and health behaviour.
Stopping Speed in ADHD · 0.62 Hedges g. In a meta-analysis of stop-signal studies, people with ADHD needed longer than controls to cancel an action, a medium effect; anxiety, autism, depression, Tourette syndrome and several other conditions showed little or no deficit.[1]
Substance Use and Addiction · 97 studies. A meta-analysis pooled 97 studies that compared heavy-use or addicted groups with healthy controls on go/no-go and stop-signal tasks. Deficits appeared for cocaine, MDMA, methamphetamine, tobacco, alcohol and gambling, and were smaller in non-dependent heavy drinkers; none appeared for opioids or cannabis, and results for internet addiction conflicted.[2]
Training and Food Intake · -0.31 Hedges g (negative = less intake). Across 32 studies of stopping practice with food cues, a single session of go/no-go training reduced later food intake or unhealthy food choice; across all training formats the pooled effect was smaller, g = -0.21.[3]
Training and Health Behaviour · 0.378 d+ (positive = improvement). Across 19 studies, inhibitory control training produced a small improvement in health behaviour, larger for go/no-go than for stop-signal training and measured mostly straight after training.[4]
Peer-reviewed sources
59
Evidence signal. The stopping deficit appears consistently at group level in ADHD and in heavy use of stimulants, alcohol and tobacco, but individual task scores are too unreliable to rank one person. Editorial judgment. The brake is real and measurable in groups; training it directly gives small effects on behaviour, measured mostly soon after training, which is why the protocol here also works on situations and habits.
02The ranking
Ranked by methodological weight
No points score was assigned.
Ranked by design quality first, then measurement precision, causal clarity and replication value.
Design quality. Controlled human data rank above observational data, which rank above animal models. Measurement precision. Objective, standardised task outcomes rank above self-report. Causal clarity. Designs that need less interpretation to support a causal reading rank higher. Replication value. Findings reproduced by independent work rank higher.
Ranking criteria, in order
03Three kinds
What each design can show
Each answers a different question.
Evidence on response inhibition comes in three kinds, and each answers a different question. Meta-analyses using a case-control design, which compares a group with a condition against a group without it, ask whether people with a diagnosis or a heavy-use history stop more slowly. Training experiments ask whether practice at stopping changes behaviour. Imaging and cohort studies ask where stopping happens and what early self-control predicts.[33]
Each kind has a ceiling. A case-control design cannot show whether slow stopping preceded a condition or followed from it. Training experiments randomise people and so allow causal claims, but mostly over short follow-up periods. Imaging shows activation, not necessity. Group differences below are reported as Hedges g or similar effect sizes, the gap between groups in standard-deviation units.
04Reliability
Groups, not individuals
The stopping literature describes groups, not individuals.
One further limit applies to all of them. Mean SSRT showed test-retest reliability, the consistency of one person's score across sessions, of .47 and .43, measured as intraclass correlation, in two studies; across seven classic tasks, reliabilities ranged from 0 to .82.[34] Tasks that separate groups well can still rank individuals poorly, so every finding below is a group-level result, not a way to score one person.[34]
The stopping literature describes groups, not individuals: task scores separate populations but are too unreliable to grade one person.
The 5 Strongest Studies on Response Inhibition
Together the five show a group-level stopping deficit in specific conditions, a broad shared brain network, a small training effect on food intake and a long-run link between childhood self-control and adult outcomes.
01Anchor
Meta-analysis of 97 case-control studies of inhibition in substance use and addiction
97 studies compared heavy-use or addicted groups with healthy controls on go/no-go commission errors and stop-signal reaction time.
This fixed-effects meta-analysis, which assumes one true effect behind the pooled studies, combined 97 studies of objective stopping tasks. It found inhibition deficits in heavy use of or dependence on cocaine, MDMA, methamphetamine, tobacco and alcohol, a smaller deficit in non-dependent heavy drinkers, and a deficit in pathological gamblers.[2] It found no deficit for opioids or cannabis, and conflicting results for internet addiction.[2]
What this proves: Weaker stopping accompanies heavy use of some substances and not others. Why it ranks first: It pools the largest number of studies, uses objective task outcomes rather than self-report, and reports its own null results. Limitation: It is case-control data, and it is only partly consistent with the stop-signal meta-analysis ranked second: the two disagree on substance dependence and pathological gambling.[1]
Rubric breakdown
The five studies, in rank order.
Ranked by design quality first, then measurement precision, causal clarity and replication value.
02
Meta-analysis of stop-signal studies across psychiatric conditions[1]
People with ADHD took longer to stop than controls (g = 0.62), as did people with OCD (g = 0.77) and schizophrenia (g = 0.69); the largest deficit was in ADHD combined with reading disability (g = 0.82).[1] Stopping was less impaired or normal in anxiety, autism, major depression, oppositional and conduct disorders, pathological gambling, reading disability, substance dependence and Tourette syndrome.[1] Proves: stopping speed separates some clinical groups from controls and not others, so it is not one trait behind all disorders. Why ranked here: One task and one latency measure give the cleanest measurement of the construct; its authors judge the OCD and schizophrenia results to need further studies.
Not scored
03
Meta-analysis of 32 go/no-go and stop-signal training studies on food intake and choice[3]
Across 32 studies, stopping practice with food cues produced a small reduction in later food intake or unhealthy choice (g = -0.21, 95% CI -0.31 to -0.11; negative values mean less intake).[3] Taken one format at a time, only single-session go/no-go training held up, and task settings varied widely.[3] Proves: in randomised comparisons, stopping practice with food cues reduces later intake by a small amount. Why ranked here: Experimental manipulation gives it the clearest causal reading in the set, but the effect is small and varies with task design.
Not scored
04
Meta-analysis of 225 functional MRI studies of response inhibition[28]
Right inferior frontal gyrus, insula, middle cingulate and paracingulate cortex and superior parietal cortex were active across interference resolution, action withholding and action cancellation, within fronto-parietal and ventral attention networks.[28] Proves: inhibition runs on a broad network shared across task types, not on a single brake region. Why ranked here: It has the widest scope of the brain evidence, but activation is correlational and cannot show that a region is necessary for stopping.
Not scored
05
Birth cohort of about 1,000 children followed to age 32, with a sibling comparison[35]
Lower childhood self-control predicted poorer physical health, more substance dependence, weaker personal finances and more criminal offending at 32, in a graded way, after controls for IQ and social class; in a second cohort of 500 sibling pairs, the sibling with lower self-control had poorer outcomes.[35] Proves: childhood self-control predicts adult health, wealth and offending; the measure is composite self-control, not response inhibition, and the design does not show cause. Why ranked here: It is observational and uses a composite measure, so it ranks last on design, but it is the only study in the set that follows consequences across decades.
Not scored
04Stakes
The Cost Shows Up in Groups, Over Years
Weaker stopping and weaker inhibition-related brain activity are associated with heavy substance use, eating outcomes and several psychiatric conditions, and broader childhood self-control predicts adult health and wealth. Most are group-level associations, not causal tests.
These associations describe risk across groups. A prospective cohort, which measures people before outcomes occur, can show that childhood self-control predicts adult outcomes. A sibling comparison, which holds family background constant, rules out some shared confounds.[35] Neither can show that improving self-control would change those outcomes.
The adolescent imaging evidence on timing is small. One study imaged 38 adolescents before heavy drinking began.[44] In another, of 80 adolescents aged 16 to 19, more left angular and supramarginal activation on no-go trials predicted more drug use 18 months later; less ventromedial prefrontal activation predicted more dependence symptoms only in the 39 baseline high-frequency users.[47] Preschool attention during waiting predicted go/no-go efficiency more than 10 years later.[48] Among nearly 60 adults, those who had waited poorly as preschoolers and still showed low self-control in their twenties and thirties were worse at suppressing responses to happy faces in their mid-forties; 26 were imaged.[49]
Weaker stopping accompanies specific conditions and heavy use of some substances at group level,[1][2] and stop-signal scores are too unreliable to explain one person's lapses.[34] Addiction itself is covered in the guide to breaking addiction.
Weaker stopping marks groups, not individuals: case-control studies find it in specific conditions, and task scores are too unreliable to read one person.
Substance Use
Heavy use of cocaine, MDMA, methamphetamine, tobacco and alcohol is associated with more commission errors or slower stopping; opioid and cannabis use showed no deficit.[2] In 38 adolescents aged 12 to 14, those who later began heavy drinking showed less no-go activation in 12 brain regions, including the rIFG, before any substantial use.[44]
What the studies measured · more presses on withhold trials, slower stop latency, weaker no-go brain activity before heavy drinking began
Eating and Weight
Obese children in residential treatment showed less inhibitory control than lean children, and the least effective inhibitors lost less weight, in a comparison of 33 obese with 31 lean children.[45] Practice at withholding responses to chocolate reduced chocolate intake in a taste test, more so in people with higher dietary restraint.[46]
What the studies measured · weaker inhibition in children in treatment, less weight lost by the weakest inhibitors, lower intake after cue-specific practice
Psychiatric Conditions
Stopping is slower in ADHD, OCD and schizophrenia than in controls, and not reliably slower in anxiety, autism, depression or Tourette syndrome.[1] The ADHD deficit has replicated across two meta-analyses, and the deficit in children did not separate ADHD from conduct disorder.[1][36]
What the studies measured · longer stop latency in ADHD, OCD and schizophrenia; little or no difference in anxiety, autism and depression
Life Course
Lower childhood self-control predicted poorer adult health, more substance dependence, weaker finances and more offending, independent of IQ and social class.[35] That measure was composite self-control, a score built from several measures rather than a stopping task, and the design is observational. In a sample of children of mothers without a college degree, each extra minute waited at age 4 predicted about 0.1 standard deviation higher achievement at 15, a modest association.[15]
What the studies measured · health, finances, dependence and offending in adulthood; school achievement at 15
05Protocol
A Four-Step Response Inhibition Protocol: Act Before the Stop Is Needed
The protocol is built from the cited studies, and no study has tested the four steps together. It works on the cue first, the plan second, the stop third and habit last.
The protocol, as a sequence.
Before the cue → When a cue is predictable → Short booster sessions → Over weeks to months
Change the Situation
Remove or distance the cue so the stop is not needed, for example by sitting away from the drinks or not having the dessert cart brought over.
Strategies that select or change the situation act early, before an impulse grows, and a review argues they may work better than resisting in the moment.[50] In daily life, personality shaped the early stages of desire, while situational factors shaped later resistance and enactment.[5]
Relying on in-the-moment resistance for a cue that is always present.
Write an If-Then Plan
For each recurring cue, write one plan in the form: if this cue appears, then I make this stopping response.
In a meta-analysis of 94 tests, if-then plans, called implementation intentions, improved goal attainment by d = 0.65 across health, academic and interpersonal goals.[51] In a two-by-two trial in dieters over four weeks, if-then plans and online food go/no-go practice each aided weight loss.[52]
Writing a general intention with no cue attached.
Practise Cue-Specific Stopping
Practise withholding a response to the exact cue you want to resist, in short go/no-go sessions.
A randomised trial of 83 adults used four 10-minute online sessions with high-energy foods as withhold cues and found weight loss and lower energy intake against control, with 97% adherence.[53] Single-session go/no-go training was the only format whose effect on food intake held on its own (g = -0.31, negative meaning less intake).[3]
Let Repetition Carry the Load
Repeat the replacement behaviour daily in the same context until it runs without effort.
In a study of simple daily behaviours, among the 39 volunteers whose curves fitted the model well, out of 82 with adequate data, the modelled time to reach 95% of peak automaticity had a median of 66 days and ranged from 18 to 254 days.[54] Across six studies with 2,274 participants, beneficial habits mediated the link between self-control and better outcomes.[55]
Expecting a fixed short deadline; the modelled times varied more than tenfold between people.
06Verdict
The verdict.
Plan for the cue and build the habit; treat stopping practice as a supplement with small effects on behaviour.
Bottom line
Knowing the rule does not guarantee the stop, so the practical aim is fewer moments that require one.
Response Inhibition Is Real, Measurable and Hard to Train Directly
Response inhibition is a stopping process whose speed can be estimated, with a broad brain network behind it. Groups with ADHD, OCD or schizophrenia stop more slowly than controls, and groups with heavy use of stimulants, alcohol or tobacco show inhibition deficits; groups with anxiety, autism or depression, and people who use opioids or cannabis, show little or no deficit. Practice at stopping reduces later food or drink intake by small amounts, measured mostly soon after training. In correlational studies, beneficial habits mediated the link between self-control and better outcomes, which points practical effort toward cues and habits.
The phrase knowing no and still acting describes a real gap, and the laboratory measures one part of it. In the stop-signal task, knowledge of the rule is complete and stopping still fails at the tracked delay when the stop process loses the race.[8][7] Whether the same race explains lapses outside the laboratory has not been tested directly.[32]
For a reader, three results carry over. Weaker stopping is a group-level association, a difference between groups that need not hold for any one member: it accompanies specific conditions,[1][2] and stop-signal task scores are too unreliable to diagnose an individual.[34] Popular accounts of willpower as a depletable resource and of preschool waiting as destiny have weakened under replication.[17][15] Food-cue stopping practice has a small pooled effect, the average across studies, on later food intake or choice.[3] On cognitive tests the overall long-term effect was not significant, although combined go/no-go and stop-signal training showed a significant long-term effect in a subgroup.[43]
No test reviewed here measures character. The tests measure a stopping process, and training it has small effects on behaviour, so the practical aim is to reduce how often that process is needed and to let habits triggered by a stable setting, or context-cued habits, carry the behaviour instead.[55][56]
Slower stopping in some groups, not all.
Stopping Is Measurable
Stopping speed can be estimated as SSRT; stopping is slower in ADHD, OCD and schizophrenia and impaired in heavy use of some substances. The measures work for groups and not for individuals.
The Brake Trains Weakly
Training effects on eating, drinking and health behaviour are small and measured mostly soon after training. Stop-signal training alone did not significantly improve cognitive performance in healthy adults.
Plan for the Cue
Change the situation, write if-then plans, practise the specific stop and repeat the replacement behaviour. The first, second and fourth steps reduce how often a fast stop is needed; the third rehearses it.
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Different forms of self-control share a neurocognitive substrate
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40
Journal
doi: 10.1093/cercor/10.3.318
Addiction, a disease of compulsion and drive: Involvement of the orbitofrontal cortex
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41
Review
doi: 10.1038/s41583-024-00807-z
From compulsivity to compulsion: The neural basis of compulsive disorders
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42
Meta
doi: 10.1016/j.appet.2015.11.013
Inhibitory control training for appetitive behaviour change: A meta-analytic investigation of mechanisms of action and moderators of effectiveness
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43
Meta
doi: 10.3389/fnins.2022.813975
Effectiveness of response inhibition training and its long-term effects in healthy adults: A systematic review and meta-analysis
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44
Journal
doi: 10.1016/j.drugalcdep.2011.06.019
Neural activation during inhibition predicts initiation of substance use in adolescence
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45
Journal
doi: 10.1016/j.eatbeh.2005.11.005
Why obese children cannot resist food: The role of impulsivity
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46
Journal
doi: 10.1016/j.appet.2010.12.017
Training inhibitory control: A recipe for resisting sweet temptations
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47
Journal
doi: 10.1016/j.addbeh.2012.07.012
Adolescents' fMRI activation to a response inhibition task predicts future substance use
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48
Journal
doi: 10.1111/j.1467-9280.2006.01732.x
Predicting cognitive control from preschool to late adolescence and young adulthood
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49
Journal
doi: 10.1073/pnas.1108561108
Behavioral and neural correlates of delay of gratification 40 years later
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50
Journal
doi: 10.1177/1745691615623247
Situational strategies for self-control
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51
Meta
doi: 10.1016/S0065-2601(06)38002-1
Implementation intentions and goal achievement: A meta-analysis of effects and processes
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52
Journal
doi: 10.1016/j.appet.2014.03.014
Targeting impulsive processes of eating behavior via the internet: Effects on body weight
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53
Journal
doi: 10.1016/j.appet.2015.06.009
Training response inhibition to food is associated with weight loss and reduced energy intake
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54
Journal
doi: 10.1002/ejsp.674
How are habits formed: Modelling habit formation in the real world
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55
Journal
doi: 10.1037/pspp0000026
More than resisting temptation: Beneficial habits mediate the relationship between self-control and positive life outcomes
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56
Journal
doi: 10.1037/0033-295X.114.4.843
A new look at habits and the habit-goal interface
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57
Journal
doi: 10.1111/j.1467-8721.2006.00435.x
Habits: A repeat performance
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58
Journal
doi: 10.1016/j.drugalcdep.2010.12.011
Resisting temptation: Decreasing alcohol-related affect and drinking behavior by training response inhibition
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59
Journal
doi: 10.1016/j.copsyc.2024.101858
Trait self-control: A Process Model perspective
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